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These discoveries suggest that individualized BP management, guided by genetic polymorphisms, endothelial reactivity, or circulating biomarkers such as endothelin-1 and nitric oxide metabolites, could optimize hemodynamic stability and advance BP-lowering strategies beyond uniform protocols 16

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radicals, will be generated in rat-heart mitochondria by both, an ubisemiquinone-dependent reaction and an iron-catalysed Haber-Weiss Cycle (Nohl et al